Med Biol Eng Comput - A nonlinear finite element simulation of balloon expandable stent for assessment of plaque vulnerability inside a stenotic artery.

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Resumo

The stresses induced on plaque wall during stent implantation inside a stenotic artery are associated with plaque rupture. The stresses in the plaque-artery-stent structure appear to be distinctly different for different plaque types in terms of both distribution and magnitude. In this study, a nonlinear finite element simulation was executed to analyze the influence of plaque composition (calcified, cellular, and hypocellular) on plaque, artery layers (intima, media, and adventitia), and stent stresses during implantation of a balloon expandable coronary stent into a stenosed artery. The atherosclerotic artery was assumed to consist of a plaque and normal arterial tissues on its outer side. The results revealed a significant influence of plaque types on the maximum stresses induced within plaque wall and artery layers during stenting, but not when calculating maximum stress on stent. The stress on stiffer calcified plaque wall was in the fracture level (2.21 MPa), whereas cellular and hypocellular plaques play a protective role by displaying less stress on their wall. The highest von Mises stresses were observed on less stiff media layer. The findings of this study suggest a lower risk of arterial vascular injury for calcified plaque, while higher risk of plaque ruptures for cellular and hypocellular plaques.

Resumo Limpo

stress induc plaqu wall stent implant insid stenot arteri associ plaqu ruptur stress plaquearteryst structur appear distinct differ differ plaqu type term distribut magnitud studi nonlinear finit element simul execut analyz influenc plaqu composit calcifi cellular hypocellular plaqu arteri layer intima media adventitia stent stress implant balloon expand coronari stent stenos arteri atherosclerot arteri assum consist plaqu normal arteri tissu outer side result reveal signific influenc plaqu type maximum stress induc within plaqu wall arteri layer stent calcul maximum stress stent stress stiffer calcifi plaqu wall fractur level mpa wherea cellular hypocellular plaqu play protect role display less stress wall highest von mise stress observ less stiff media layer find studi suggest lower risk arteri vascular injuri calcifi plaqu higher risk plaqu ruptur cellular hypocellular plaqu

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