Med Biol Eng Comput - Coronary-aortic interaction during ventricular isovolumic contraction.

Tópicos

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Resumo

In earlier work, we suggested that the start of the isovolumic contraction period could be detected in arterial pressure waveforms as the start of a temporary pre-systolic pressure perturbation (AIC(start), start of the Arterially detected Isovolumic Contraction), and proposed the retrograde coronary blood volume flow in combination with a backwards traveling pressure wave as its most likely origin. In this study, we tested this hypothesis by means of a coronary artery occlusion protocol. In six Yorkshire ? Landrace swine, we simultaneously occluded the left anterior descending (LAD) and left circumflex (LCx) artery for 5 s followed by a 20-s reperfusion period and repeated this sequence at least two more times. A similar procedure was used to occlude only the right coronary artery (RCA) and finally all three main coronary arteries simultaneously. None of the occlusion protocols caused a decrease in the arterial pressure perturbation in the aorta during occlusion (P > 0.20) nor an increase during reactive hyperemia (P > 0.22), despite a higher deceleration of coronary blood volume flow (P = 0.03) or increased coronary conductance (P = 0.04) during hyperemia. These results show that the pre-systolic aortic pressure perturbation does not originate from the coronary arteries.

Resumo Limpo

earlier work suggest start isovolum contract period detect arteri pressur waveform start temporari presystol pressur perturb aicstart start arteri detect isovolum contract propos retrograd coronari blood volum flow combin backward travel pressur wave like origin studi test hypothesi mean coronari arteri occlus protocol six yorkshir landrac swine simultan occlud left anterior descend lad left circumflex lcx arteri s follow s reperfus period repeat sequenc least two time similar procedur use occlud right coronari arteri rca final three main coronari arteri simultan none occlus protocol caus decreas arteri pressur perturb aorta occlus p increas reactiv hyperemia p despit higher deceler coronari blood volum flow p increas coronari conduct p hyperemia result show presystol aortic pressur perturb origin coronari arteri

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